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• Because D 1 dopamine receptors are known to act through stimulatory heterotrimeric guanosine triphosphate-binding (G s ) proteins and activation of the cAMP pathway, up-regulation of this pathway in the nucleus accumbens could account for the functional supersensitivity of D 1 receptors observed in these neurons--which occurs in the absence of detectable changes in the receptors themselves--after chronic cocaine (or other stimulant) exposure (18 ).
• In the next generation seeds were screened for ABA supersensitivity .
• The effects of inactivity on new receptor synthesis and denervation supersensitivity are mediated by Ca 2 influx (24 ).